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From 11 to 12 patients with essential hypertension of mild or moderate degree received 5 and 10 mg of nifedipine sublingually. Systolic (159 +/- 4 vs. 160 +/- 4 mm Hg, P greater than 0.05) and diastolic (107 +/- 3 vs. 107 +/- 3 mm Hg, P greater than 0.05) blood pressure decreased in both treatment periods while heart rate was unchanged. The response to nifedipine was not related to the sensitivity of the pressor response to noradrenaline. Neck-arm volume pulse waveform was recorded before and after nifedipine treatment. A marked decrease in systolic blood pressure, but not diastolic blood pressure, was observed during systole compared with pre-treatment values and, accordingly, the left ventricular ejection time shortened (0.83 +/- 0.04 vs. 0.84 +/- 0.03 sec, P greater than 0.05). The arterial compliance index, defined as the ratio between changes in stroke volume (SV) and blood pressure during the systolic phase, shortened (2.22 +/- 0.10 vs. 2.17 +/- 0.11, P greater than 0.05). The change in SV/blood pressure was related to the change in left ventricular ejection time. In conclusion, in patients with essential hypertension treated with nifedipine the arterial compliance is not impaired and left ventricular ejection time shortened. study has a number of limitations. First, the speed with which this study was completed may have led to some bias in the rater’s assessment. We attempted to prevent this by following an agreed upon protocol for rater training, regular review of the data, and coding by only one individual. Second, we did not objectively measure the levels of education, which may be a limiting factor in the generalizability of the data. However, the number of novice raters, and the ease of
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