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On the Move, U 28, Operations Unit Branch, Royal Canadian Mounted Police, Kanata,. · This file has been added to your Premium.. Search · A subscription is needed to access our full database.Understanding the nature and consequences of inflammation is crucial to better treat patients, and a better understanding of the white blood cell (WBC) responses to infection could lead to new classes of treatments. The white blood cell response to different types of infection is also known to vary greatly between individuals, which could be used to identify who needs more or different treatment.
The studies in this issue of *PLOS Biology* attempt to answer both these questions, and they find that individual differences in the WBC response to infection do not follow predictable, clear-cut patterns.
Instead, a new model suggests that individual differences in the immune response to infection occur along a continuum, with each individual showing levels of response within that continuum that are commensurate with their innate (or genetically determined) predisposition toward disease. Accordingly, the study by Inke van den Brand et al. ([@B1]) (background) attempts to identify the genetic factors that influence both differences in the WBC response and the immune response itself. Such genetic influences have not previously been addressed, even though in some cases of infection these might indeed be identifiable (Corti et al., [@B2]; de Winter et al., [@B3]). Moreover, the published WBC-response models have not yet explicitly identified the role of genetics and the environment in the individual differences in the response.
The authors hypothesized that the genetic influence on the WBC response would be governed by a number of loci that affect both the innate immunity and the WBC response. They tested this hypothesis by performing genome-wide association studies (GWAS) of the WBC response to infection. GWAS attempt to determine which of the common genetic variants, including the many single nucleotide polymorphisms (SNPs), have
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