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Chlamydia trachomatis serovar L1 is a gram-negative obligate intracellular bacterium that causes abortion and stillbirths in pregnant women and premature labor in neonates. This study was undertaken to examine the role of nuclear factor-kappaB (NF-kappaB) in cytokine expression and production in the early stages of infection of human placental choriocarcinoma cells (JAR) with C. trachomatis serovar L1. NF-kappaB activation and various cytokine mRNA levels were determined by immunoblot and reverse transcription-polymerase chain reaction (RT-PCR) assays, respectively. Elispot and enzyme-linked immunosorbent assay (ELISA) assays were used to determine the production of tumor necrosis factor-alpha (TNF-alpha), interleukin-1beta (IL-1beta), IL-6, and IL-8, respectively. C. trachomatis serovar L1 infection induced a marked increase in the production of IL-1beta and IL-8, but not TNF-alpha, in JAR cells. NF-kappaB was activated at 5 min post
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